Physcian Enquiries

Wave Brilliance Australia is  a distributor and supplier of the ExMI pelvic floor rehabilitation system in Australia and New Zealand, a corporate member of the Continence Foundation of Australia, R.A.C.G.P. Accredited Provider and member of the Prostate Cancer Foundation of Australia.

Our Mission Statement - A vision to see, the power to treat

Empowering GP's to treat their patients for disorders of the pelvic floor

 

At last, a highly effective, affordable treatment option is available at primary care level. We encourage all GP's to evaluate the benefits of ExMI and include this approach into their practice.


The Role of Extracorporeal Magnetic Innervation (ExMI™)

In the Treatment of Urinary Incontinence

 

Class Type Patho-physiology Functional Defect or Level of Severity Does ExMI have a role?

Decreased Urethral Resistance

Anatomic Support Defects (GSI I/II)

Failure of adequate urethral closure

Support Defect

Yes

 

(Stress)

 

Greater forces on posterior vs. anterior wall of urethra

Anatomic Defect

No

     

Functional Support

Yes

 

Intrinsic Sphincter Deficiency (ISD)

Deficiency in urethral closure mechanism

Mild

Yes

     

Moderate

Yes

     

Severe

Probably Not

 

Failure to Inhibit the Detrusor

Failure to contract pelvic floor releases detrusor reflex

Neurogenic – Mild / Moderate

Yes

     

Neurogenic – Severe

No

     

Behavioral

Yes

     

Mechanical

Probably Not

Increased Urethral Resistance (Overflow / Retention)

Anatomic Obstruction

Urethral compression or narrowing

 

No

 

Functional Obstruction

Failure of normal relaxation

Neurogenic

Yes

     

Behavioral (failure to relax external sphincter)

Yes

 

Inhibition of Detrusor Activity

Failure to relax pelvic floor muscles inhibits detrusor ability to start and maintain a contraction

Neurogenic

Yes

     

Behavioral

Yes

     

Situational

Yes

Increased Intravesical Pressure (Urge)

Uninhibited Detrusor Contractions

Increased intravesical pressure overcomes urethral resistance

Overactive Bladder

Yes

     

Detrusor Hyper Reflexia

Yes

   

Decreased accommodation

Fibrosis

No

     

Neurogenic

No

Decreased Intravesical Pressure (Overflow / Retention)

Peripheral Denervation or Neuropathy

Absent neuro input

Congenital

No

     

Diabetics

Perhaps

 

Detrusor Myopathy

Smooth muscle damage

Fibrosis

No

     

Inflammation

No